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a(MESH:"alpha-conotoxin MII")

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
alpha-conotoxin MII
Namespace
mesh
Namespace Version
20181007
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/8ccfed235e418e4c8aa576f9a5ef0f838e794c7f/external/mesh-names.belns

Appears in Networks 2

NACHO Mediates Nicotinic Acetylcholine Receptor Function throughout the Brain v1.0.0

Naturally-expressed nicotinic acetylcholine receptor subtypes v1.0.0

In-Edges 0

Out-Edges 2

a(MESH:"alpha-conotoxin MII") decreases complex(a(CHEBI:epibatidine), a(MESH:"alpha6beta2 nicotinic acetylcholine receptor")) View Subject | View Object

This NACHO-mediated [3H]epibatidine binding to alpha6beta2 transfectants was also displaced by conotoxin MII (Figure 4E) PubMed:28445721

Appears in Networks:

a(MESH:"alpha-conotoxin MII") decreases act(a(HBP:"alpha-3 beta-2 nAChR")) View Subject | View Object

Initially, a-CtxMII was thought to be highly selective for a3b2*-nAChRs [124] and became a useful tool for investigating receptor structure [125]. PubMed:21787755

Appears in Networks:

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.