The node-based overlap between this network and other networks is calculated as the Szymkiewicz-Simpson coefficient of their respective nodes. Up to the top 10 are shown below.
|Increased NF-κB signalling up-regulates BACE1 expression and its therapeutic potential in Alzheimer's disease. v1.0.0||65%|
|Estrogen receptor-α is localized to neurofibrillary tangles in Alzheimer's disease v1.0.0||50%|
|Alzheimer's disease pathological lesions activate the spleen tyrosine kinase. v1.0.0||35%|
|Anatabine lowers Alzheimer's Aβ production in vitro and in vivo v1.0.0||35%|
|Chronic Anatabine Treatment Reduces Alzheimer ’ s Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of AD v1.0.0||35%|
|Phytochemicals as inhibitors of NF-κB for treatment of Alzheimer’s disease v1.0.0||33%|
|Identification of a novel aspartic protease (Asp 2) as beta-secretase v1.0.0||30%|
|Anti-inflammatory activity of anatabine via inhibition of STAT3 phosphorylation v1.0.0||28%|
|Anatabine ameliorates experimental autoimmune thyroiditis. v1.0.0||27%|
|Tau Modifications v1.9.5||25%|
BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.
If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.