The node-based overlap between this network and other networks is calculated as the Szymkiewicz-Simpson coefficient of their respective nodes. Up to the top 10 are shown below.
|Increased NF-κB signalling up-regulates BACE1 expression and its therapeutic potential in Alzheimer's disease. v1.0.0||50%|
|Anti-inflammatory activity of anatabine via inhibition of STAT3 phosphorylation v1.0.0||40%|
|Anatabine lowers Alzheimer's Aβ production in vitro and in vivo v1.0.0||40%|
|Selective activation of α7 nicotinic acetylcholine receptor by PHA-543613 improves Aβ25-35-mediated cognitive deficits in mice v1.0.0||36%|
|Chronic Anatabine Treatment Reduces Alzheimer ’ s Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of AD v1.0.0||35%|
|Upstream regulators and downstream effectors of NF-κBinAlzheimer's disease v1.0.0||33%|
|Anatabine ameliorates experimental autoimmune thyroiditis. v1.0.0||33%|
|Effects of peptides derived from BACE1 catalytic domain on APP processing v1.0.0||30%|
|Inflammasome activation and innate immunity in Alzheimer’s disease v1.0.2||29%|
|Inﬂammasome Involvement in Alzheimer’s Disease v1.0.0||28%|
BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.
If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.