path(MESH:Thrombosis) association path(MESH:Thalassemia)

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PubMed:25307023

Multiple haemolytic disorders and therapeutic interventions produce substantial intravascular haemolysis. Examples include PNH, SCD, thalassaemias, glucose-6-phosphate dehydrogenase (G6PD) deficiency, hereditary spherocytosis and stomatocytosis, pyruvate kinase deficiency, autoimmune haemolytic anaemia, microangiopathies, acute haemolytic transfusion reactions, mechanical circulatory support [e.g., left ventricular assist device (LVAD)/extracorporeal membrane oxygenation (ECMO)], RBC transfusions and infusions of RBC substitutes. These disorders, therapies and procedures are also associated with an increased risk of thrombosis.

Related Edges 34

• a(HM:"Glucose-6-phosphate dehydrogenase deficiency") increases path(MESH:Hemolysis)
• a(HM:"Glucose-6-phosphate dehydrogenase deficiency") association path(MESH:Thrombosis)
• a(HM:"acute hemolytic transfusion reaction") association path(MESH:Thrombosis)
• a(MESH:"Blood Substitutes") increases path(MESH:Hemolysis)
• a(MESH:"Blood Substitutes") association path(MESH:Thrombosis)
• a(MESH:"Blood Transfusion") increases path(MESH:Hemolysis)
• a(MESH:"Blood Transfusion") association path(MESH:Thrombosis)
• path(HP:"Paroxysmal nocturnal hemoglobinuria") increases path(MESH:Hemolysis)
• path(HP:"Paroxysmal nocturnal hemoglobinuria") association path(MESH:Thrombosis)
• path(HP:"autoimmune hemolytic anemia") increases path(MESH:Hemolysis)
• path(HP:"autoimmune hemolytic anemia") association path(MESH:Thrombosis)
• path(HP:Stomatocytosis) increases path(MESH:Hemolysis)
• path(HP:Stomatocytosis) association path(MESH:Thrombosis)
• path(MESH:"Anemia, Sickle Cell") increases path(MESH:Hemolysis)
• path(MESH:"Anemia, Sickle Cell") association path(MESH:Thrombosis)
• path(MESH:"Pyruvate Kinase Deficiency of Red Cells") increases path(MESH:Hemolysis)
• path(MESH:"Pyruvate Kinase Deficiency of Red Cells") association path(MESH:Thrombosis)
• path(MESH:"Spherocytosis, Hereditary") increases path(MESH:Hemolysis)
• path(MESH:"Spherocytosis, Hereditary") association path(MESH:Thrombosis)
• path(MESH:"Thrombotic Microangiopathies") increases path(MESH:Hemolysis)
• path(MESH:"Thrombotic Microangiopathies") association path(MESH:Thrombosis)
• path(MESH:Thalassemia) increases path(MESH:Hemolysis)
• path(MESH:Thalassemia) association path(MESH:Thrombosis)
• path(MESH:Thrombosis) association path(HP:"Paroxysmal nocturnal hemoglobinuria")
• path(MESH:Thrombosis) association path(MESH:"Anemia, Sickle Cell")
• path(MESH:Thrombosis) association a(HM:"Glucose-6-phosphate dehydrogenase deficiency")
• path(MESH:Thrombosis) association path(MESH:"Spherocytosis, Hereditary")
• path(MESH:Thrombosis) association path(HP:Stomatocytosis)
• path(MESH:Thrombosis) association path(MESH:"Pyruvate Kinase Deficiency of Red Cells")
• path(MESH:Thrombosis) association path(HP:"autoimmune hemolytic anemia")
• path(MESH:Thrombosis) association path(MESH:"Thrombotic Microangiopathies")
• path(MESH:Thrombosis) association a(HM:"acute hemolytic transfusion reaction")
• path(MESH:Thrombosis) association a(MESH:"Blood Transfusion")
• path(MESH:Thrombosis) association a(MESH:"Blood Substitutes")

Cell Ontology (CL)

macrophage

MeSH

Plasma

MeSH

Urine

Text Location

Review

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If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.

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