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Entity

Name
Receptors, Nicotinic
Namespace
mesh
Namespace Version
20180906
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/b46b65c3da259b6e86026514dfececab7c22a11b/external/mesh-names.belns

Appears in Networks 1

In-Edges 1

Out-Edges 1

p(MESH:"Receptors, Nicotinic", pmod(Ph)) increases act(p(MESH:Agrin)) View Subject | View Object

In nAChRs, several phosphorylation sites (104) that control desensitization in mus- cle and 􏰀7-nAChR also contribute to end plate localization by agrin-induced tyrosine phosphorylation of the cytoskeletal protein 43K-rapsyn PubMed:23038257

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.