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a(CHEBI:berberine)

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
berberine
Namespace
chebi
Namespace Version
20190224
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/c328ad964c08967a0417a887510b97b965a62fa5/external/chebi-names.belns

Appears in Networks 1

Phytochemicals as inhibitors of NF-κB for treatment of Alzheimer’s disease v1.0.0

In-Edges 0

Out-Edges 7

a(CHEBI:berberine) decreases path(MESH:Inflammation) View Subject | View Object

Berberine suppressed inflammatory events occurring in several inflammation-related diseases [186,187]. PubMed:29179999

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a(CHEBI:berberine) decreases p(HGNC:RELA, pmod(Ph)) View Subject | View Object

Furthermore, berberine decreased the phosphorylation and expression of p65 [188]. PubMed:29179999

Appears in Networks:

a(CHEBI:berberine) decreases p(HGNC:RELA) View Subject | View Object

Furthermore, berberine decreased the phosphorylation and expression of p65 [188]. PubMed:29179999

Appears in Networks:

a(CHEBI:berberine) decreases p(HGNC:NFKBIA, pmod(Ph)) View Subject | View Object

Also, berberine inhibited the phosphorylation of IB. PubMed:29179999

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a(CHEBI:berberine) decreases act(p(HGNC:MAPK14)) View Subject | View Object

Berberine inhibited the p38,ERK and Akt signaling pathways, which were stimulated by A PubMed:29179999

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a(CHEBI:berberine) decreases act(p(FPLX:ERK)) View Subject | View Object

Berberine inhibited the p38,ERK and Akt signaling pathways, which were stimulated by A PubMed:29179999

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a(CHEBI:berberine) decreases act(p(FPLX:AKT)) View Subject | View Object

Berberine inhibited the p38,ERK and Akt signaling pathways, which were stimulated by A PubMed:29179999

Appears in Networks:

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.