TIAM1

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name: TIAM1
Namespace: hgnc
Namespace Version: 20180906
Namespace URL: https://raw.githubusercontent.com/pharmacome/terminology/b46b65c3da259b6e86026514dfececab7c22a11b/external/hgnc-names.belns

Appears in Networks 2

Tau Modifications v1.9.5

Tau Modifications Sections of NESTOR

Nuclear Factor Kappa-light-chain-enhancer of Activated B Cells (NF-κB) - a Friend, a Foe, or a Bystander - in the Neurodegenerative Cascade and Pathogenesis of Alzheimer's Disease v1.0.0

In-Edges 1

complex(GO:"NF-kappaB complex") regulates p(HGNC:TIAM1) View Subject | View Object

Furthermore, NF-κB directly regulates the expression of a plethora of cell adhesion molecules such as neural cell adhesion molecule (NCAM) [202], slit and Trk-like family member 1 (SLITRK1) [203], glial cell-derived neurotrophic factor receptor α1 (GFRα1) [204], and T-lymphoma and metastasis 1 (TLAM1) [203], key players in directing axon growth. PubMed:28745240

Appears in Networks:

Nuclear Factor Kappa-light-chain-enhancer of Activated B Cells (NF-κB) - a Friend, a Foe, or a Bystander - in the Neurodegenerative Cascade and Pathogenesis of Alzheimer's Disease v1.0.0

Annotations

MeSH: Alzheimer Disease

Out-Edges 2

p(HGNC:TIAM1) hasVariant p(HGNC:TIAM1, pmod(Ph)) View Subject | View Object

Appears in Networks:

Tau Modifications v1.9.5

p(HGNC:TIAM1) increases a(GO:axon) View Subject | View Object

Appears in Networks:

Nuclear Factor Kappa-light-chain-enhancer of Activated B Cells (NF-κB) - a Friend, a Foe, or a Bystander - in the Neurodegenerative Cascade and Pathogenesis of Alzheimer's Disease v1.0.0

Annotations

MeSH: Alzheimer Disease

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If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.