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  3. path(MESH:"Muscular Atrophy")

path(MESH:"Muscular Atrophy")

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
Muscular Atrophy
Namespace
MeSH
Namespace Version
20181007
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/01c9daa61012b37dd0a1bc962521ba51a15b38f1/external/mesh-names.belns

Appears in Networks 1

Axonal Transport, Tau Protein, and Neurodegeneration in Alzheimer’s Disease v1.0.0

In-Edges 1

p(HBP:"4R tau") increases path(MESH:"Muscular Atrophy") View Subject | View Object

TSubsequently, Wallerian degeneration and severe muscle wasting and motoric problems demon- strated the extensive neurodegeneration caused by the overexpression of human tau protein in a gene- dosage fashion. PubMed:12428809

Appears in Networks:

Out-Edges 0

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.