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Neuromuscular Junction

Name
Neuromuscular Junction
Namespace Keyword
MeSHAnatomy
Namespace
MeSH
Namespace Version
20170511
Namespace URL
https://arty.scai.fraunhofer.de/artifactory/bel/annotation/mesh-anatomy/mesh-anatomy-20170511.belanno

Sample Annotated Edges 2

p(HGNC:ACHE) decreases act(a(CHEBI:nicotine)) View Subject | View Object

At the neuromuscular junction, nicotinic function is enhanced by inhibition of acetylcholinesterase (AChE), the enzyme that metabolizes the endogenous neurotransmitter ACh. PubMed:19126755

Appears in Networks:
Annotations
MeSH
Neuromuscular Junction
Text Location
Review

p(HGNC:BAIAP3) regulates bp(GO:"retrograde trans-synaptic signaling") View Subject | View Object

AEX-1, predominantly expressed in muscles and intestine, regulates the retrograde signaling at neuromuscular junctions and is required for the normal localization of synaptic vesicle fusion protein UNC-13 PubMed:29191965

Appears in Networks:
Annotations
MeSH
Neuromuscular Junction

Showing 2 of 2 edges

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.