p(HGNC:SQSTM1) increases bp(GO:autophagy)

View Subject View Object

PubMed:18930136

Recent models propose that p62 and HDAC6 function analogously to facilitate autophagic degradation of proteins that display specific polyubiquitin topology.

Related Edges 3

• p(HGNC:HDAC6) increases bp(GO:autophagy)
• p(HGNC:HDAC6) increases bp(GO:"ubiquitin-dependent protein catabolic process")
• p(HGNC:SQSTM1) increases bp(GO:"ubiquitin-dependent protein catabolic process")

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.

---