Versions 6

Provenance

Upload
aparna.mohan@causalitybiomodels.com at 2018-12-11 07:05:39
Authors
Aparna
Contact
aparna.mohan@causalitybiomodels.com
Description
CMB curation project: pilot_16628
License
Private Ownership to Fraunhofer Institute SCAI
Copyright
Copyright (c) 2018 Fraunhofer Institute SCAI, All rights reserved
Number Nodes
3
Number Edges
4
Number Components
1
Network Density
0.666667
Average Degree
1.33333
Number Citations
1
Number BEL Errors
51

Content Statistics

Network Overlap

The node-based overlap between this network and other networks is calculated as the Szymkiewicz-Simpson coefficient of their respective nodes. Up to the top 10 are shown below.

Network Overlap
BEL Framework Large Corpus Document v20170611 33%
Neurotransmitter and Insulin NeuroMMSig enrichments v0.0.1 33%

Sample Edges

p(MGI:Mapt) association path(MeSHDisease:"Alzheimer Disease")

Agents capable of preventing the misfolding and sequestration of the microtubule-stabilizing protein tau into insoluble fibrillar aggregates hold considerable promise for the prevention and/or treatment of neurodegenerative tauopathies such as Alzheimer's disease PubMed:20392114

Annotations
MeSH
Central Nervous System

p(MGI:Mapt) association path(MeSHDisease:Amyloidosis)

Agents capable of preventing the misfolding and sequestration of the microtubule-stabilizing protein tau into insoluble fibrillar aggregates hold considerable promise for the prevention and/or treatment of neurodegenerative tauopathies such as Alzheimer's disease PubMed:20392114

Annotations
MeSH
Central Nervous System

path(MeSHDisease:"Alzheimer Disease") association p(MGI:Mapt)

Agents capable of preventing the misfolding and sequestration of the microtubule-stabilizing protein tau into insoluble fibrillar aggregates hold considerable promise for the prevention and/or treatment of neurodegenerative tauopathies such as Alzheimer's disease PubMed:20392114

Annotations
MeSH
Central Nervous System

path(MeSHDisease:Amyloidosis) association p(MGI:Mapt)

Agents capable of preventing the misfolding and sequestration of the microtubule-stabilizing protein tau into insoluble fibrillar aggregates hold considerable promise for the prevention and/or treatment of neurodegenerative tauopathies such as Alzheimer's disease PubMed:20392114

Annotations
MeSH
Central Nervous System

Sample Nodes

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of the open source project, PyBEL. Please feel free to contact us here to give us feedback or report any issues.